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rabbit anti rat txnip  (Thermo Fisher)


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    Structured Review

    Thermo Fisher rabbit anti rat txnip
    The expression of TNXIP in the lesioned spinal cord following compression was observed. ( A ) The TNXIP/NeuN expression in the anterior horn of lesioned spinal cords was analyzed by immunofluorescence. The mean fluorescence intensities of <t>TXNIP</t> were analyzed by Image J software, and represented as mean gray values. ( B ) The TXNIP expression in the lesioned spinal cords was analyzed by Western blot. The gray values of protein bands were analyzed by Image J software, and protein expression was normalized to GAPDH. *P<0.05, **P<0.01.
    Rabbit Anti Rat Txnip, supplied by Thermo Fisher, used in various techniques. Bioz Stars score: 94/100, based on 7 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
    https://www.bioz.com/result/rabbit anti rat txnip/product/Thermo Fisher
    Average 94 stars, based on 7 article reviews
    rabbit anti rat txnip - by Bioz Stars, 2026-03
    94/100 stars

    Images

    1) Product Images from "TXNIP Participated in NLRP3-Mediated Inflammation in a Rat Model of Cervical Spondylotic Myelopathy"

    Article Title: TXNIP Participated in NLRP3-Mediated Inflammation in a Rat Model of Cervical Spondylotic Myelopathy

    Journal: Journal of Inflammation Research

    doi: 10.2147/JIR.S373614

    The expression of TNXIP in the lesioned spinal cord following compression was observed. ( A ) The TNXIP/NeuN expression in the anterior horn of lesioned spinal cords was analyzed by immunofluorescence. The mean fluorescence intensities of TXNIP were analyzed by Image J software, and represented as mean gray values. ( B ) The TXNIP expression in the lesioned spinal cords was analyzed by Western blot. The gray values of protein bands were analyzed by Image J software, and protein expression was normalized to GAPDH. *P<0.05, **P<0.01.
    Figure Legend Snippet: The expression of TNXIP in the lesioned spinal cord following compression was observed. ( A ) The TNXIP/NeuN expression in the anterior horn of lesioned spinal cords was analyzed by immunofluorescence. The mean fluorescence intensities of TXNIP were analyzed by Image J software, and represented as mean gray values. ( B ) The TXNIP expression in the lesioned spinal cords was analyzed by Western blot. The gray values of protein bands were analyzed by Image J software, and protein expression was normalized to GAPDH. *P<0.05, **P<0.01.

    Techniques Used: Expressing, Immunofluorescence, Fluorescence, Software, Western Blot

    Inhibition of TXNIP improved CSM-induced behavioral deficits. ( A ) The21-point Basso, Beattie and Bresnahan (BBB) locomotor rating scale; ( B ) cold allodynia; ( C ) inclined plane test. Compared with the sham group, **P<0.01; compared with the scrambled group, # P<0.05, ## P<0.01.
    Figure Legend Snippet: Inhibition of TXNIP improved CSM-induced behavioral deficits. ( A ) The21-point Basso, Beattie and Bresnahan (BBB) locomotor rating scale; ( B ) cold allodynia; ( C ) inclined plane test. Compared with the sham group, **P<0.01; compared with the scrambled group, # P<0.05, ## P<0.01.

    Techniques Used: Inhibition

    Inhibition of TXNIP reduced apoptosis in the anterior horn of the lesioned area following CSM. ( A ) Positive TUNEL staining control. ( B ) Apoptosis in the anterior horn of the lesioned area was analyzed by TUNEL staining. Black arrows show apoptotic cells. Apoptosis rate (%) = the numbers of apoptosis cells/total numbers of cells ×100%. The numbers of cells were counted using Image J software. Compared with the sham group, **P<0.01; compared with the scrambled group, ## P<0.01.
    Figure Legend Snippet: Inhibition of TXNIP reduced apoptosis in the anterior horn of the lesioned area following CSM. ( A ) Positive TUNEL staining control. ( B ) Apoptosis in the anterior horn of the lesioned area was analyzed by TUNEL staining. Black arrows show apoptotic cells. Apoptosis rate (%) = the numbers of apoptosis cells/total numbers of cells ×100%. The numbers of cells were counted using Image J software. Compared with the sham group, **P<0.01; compared with the scrambled group, ## P<0.01.

    Techniques Used: Inhibition, TUNEL Assay, Staining, Control, Software

    Inhibition of TXNIP declined the TXNIP expression in the anterior horn of the lesioned area following CSM. The mean fluorescence intensities of TXNIP/NeuN were analyzed by immunofluorescence. The mean gray values were analyzed using Image J software. Compared with the sham group, *P<0.05, **P<0.01; compared with the scrambled group, ## P<0.01.
    Figure Legend Snippet: Inhibition of TXNIP declined the TXNIP expression in the anterior horn of the lesioned area following CSM. The mean fluorescence intensities of TXNIP/NeuN were analyzed by immunofluorescence. The mean gray values were analyzed using Image J software. Compared with the sham group, *P<0.05, **P<0.01; compared with the scrambled group, ## P<0.01.

    Techniques Used: Inhibition, Expressing, Fluorescence, Immunofluorescence, Software

    Inhibition of TXNIP declined the NLRP3 expression in the anterior horn of the lesioned area following CSM. The mean fluorescence intensities of NLRP3/NeuN were analyzed by immunofluorescence. The mean gray values were analyzed by Image J software. Compared with the sham group, **P<0.01; compared with the scrambled group, ## P<0.01.
    Figure Legend Snippet: Inhibition of TXNIP declined the NLRP3 expression in the anterior horn of the lesioned area following CSM. The mean fluorescence intensities of NLRP3/NeuN were analyzed by immunofluorescence. The mean gray values were analyzed by Image J software. Compared with the sham group, **P<0.01; compared with the scrambled group, ## P<0.01.

    Techniques Used: Inhibition, Expressing, Fluorescence, Immunofluorescence, Software

    Inhibition TXNIP declined the NLRP3 mediated pro-caspase-1 expression in the anterior horn of the lesioned area following CSM. ( A ) The mean fluorescence intensities of pro-caspase-1/NeuN were analyzed by immunofluorescence. ( B ) The expression levels of TXNIP, NLRP3 and pro-caspase-1 in the lesioned spinal cords were measured by Western blot. The mean grays were analyzed by the Image J software. Compared with the sham group, **P<0.01; compared with the scrambled group, ## P<0.01.
    Figure Legend Snippet: Inhibition TXNIP declined the NLRP3 mediated pro-caspase-1 expression in the anterior horn of the lesioned area following CSM. ( A ) The mean fluorescence intensities of pro-caspase-1/NeuN were analyzed by immunofluorescence. ( B ) The expression levels of TXNIP, NLRP3 and pro-caspase-1 in the lesioned spinal cords were measured by Western blot. The mean grays were analyzed by the Image J software. Compared with the sham group, **P<0.01; compared with the scrambled group, ## P<0.01.

    Techniques Used: Inhibition, Expressing, Fluorescence, Immunofluorescence, Western Blot, Software



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    Thermo Fisher rabbit anti rat txnip
    The expression of TNXIP in the lesioned spinal cord following compression was observed. ( A ) The TNXIP/NeuN expression in the anterior horn of lesioned spinal cords was analyzed by immunofluorescence. The mean fluorescence intensities of <t>TXNIP</t> were analyzed by Image J software, and represented as mean gray values. ( B ) The TXNIP expression in the lesioned spinal cords was analyzed by Western blot. The gray values of protein bands were analyzed by Image J software, and protein expression was normalized to GAPDH. *P<0.05, **P<0.01.
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    Abcam rabbit anti rat txnip
    The impact of ROS and NLRP3 inflammasome on the expression of pyroptosis associated gene and protein. (A)The gene and protein expression of <t>TXNIP,</t> NLRP3, and Caspae-1 in NRK-52E cells under 200 μg/ml uric acid stimulation or not for 24 hr detected by Real-time PCR and western blot techniques (n=3). The cells were categorized into four groups: the control group (0 μg/ml uric acid), the UA group (200 μg/ml uric), the UA+ROS (−) group (200 μg/ml uric + the ROS inhibitors NAC), as well as the UA+mtROS (−) group (200 μg/ml uric+ mtROS inhibitors mt-TEMPO). Data were expressed as mean ± SD. Compared with the UA group, ** P < 0.0001, * P < 0.05. Compared with the UA+ROS (−) group, ⋆⋆ P < 0.0001, ⋆ P < 0.05. Compared with UA+mtROS(−) group, # # P < 0.0001, # P < 0.05.(B) The pyroptosis percent in NRK-52E cells subjected to 200 μg/ml uric acid or not for 24 hr via flow cytometry method (n=3). The cells were divided into four groups: the control group (0 μg/ml uric acid), the UA group (200 μg/ml uric), the UA+NLRP3 (−) group (200 μg/ml uric + the NLRP3 inhibitors MCC950), <t>the</t> <t>UA+Caspase-1(−)</t> group (200 μg/ml uric+ Caspase-1 inhibitors Z-YVAD-FMK). Data were expressed as mean ± SD. Compared with the UA group, ** P < 0.0001, * P < 0.05. Compared with the UA+NLRP3 (-) group, ⋆⋆ P < 0.0001, ⋆ P < 0.05. Compared with UA+Caspase-1(−) group, ## P < 0.0001, # P < 0.05.(C) The gene and protein expression of NLRP3, ASC, Caspae-1, Activated-Caspase-1, GSDMD, IL-1β, and IL-18 in NRK-52E cells when exposed to 200 μg/ml uric acid or not for 24 hr using Real-time PCR and western blot method (n=3).
    Rabbit Anti Rat Txnip, supplied by Abcam, used in various techniques. Bioz Stars score: 98/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
    https://www.bioz.com/result/rabbit anti rat txnip/product/Abcam
    Average 98 stars, based on 1 article reviews
    rabbit anti rat txnip - by Bioz Stars, 2026-03
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    Image Search Results


    The expression of TNXIP in the lesioned spinal cord following compression was observed. ( A ) The TNXIP/NeuN expression in the anterior horn of lesioned spinal cords was analyzed by immunofluorescence. The mean fluorescence intensities of TXNIP were analyzed by Image J software, and represented as mean gray values. ( B ) The TXNIP expression in the lesioned spinal cords was analyzed by Western blot. The gray values of protein bands were analyzed by Image J software, and protein expression was normalized to GAPDH. *P<0.05, **P<0.01.

    Journal: Journal of Inflammation Research

    Article Title: TXNIP Participated in NLRP3-Mediated Inflammation in a Rat Model of Cervical Spondylotic Myelopathy

    doi: 10.2147/JIR.S373614

    Figure Lengend Snippet: The expression of TNXIP in the lesioned spinal cord following compression was observed. ( A ) The TNXIP/NeuN expression in the anterior horn of lesioned spinal cords was analyzed by immunofluorescence. The mean fluorescence intensities of TXNIP were analyzed by Image J software, and represented as mean gray values. ( B ) The TXNIP expression in the lesioned spinal cords was analyzed by Western blot. The gray values of protein bands were analyzed by Image J software, and protein expression was normalized to GAPDH. *P<0.05, **P<0.01.

    Article Snippet: The primary antibodies included rabbit anti-rat TXNIP (1:800, MA5-32771, ThermoFisher), NLRP3 (1:800, MA5-32255, ThermoFisher), pro-caspase-1 (1:800, MA5-35833, ThermoFisher) and GAPDH (1:10000, PA1-16777, ThermoFisher).

    Techniques: Expressing, Immunofluorescence, Fluorescence, Software, Western Blot

    Inhibition of TXNIP improved CSM-induced behavioral deficits. ( A ) The21-point Basso, Beattie and Bresnahan (BBB) locomotor rating scale; ( B ) cold allodynia; ( C ) inclined plane test. Compared with the sham group, **P<0.01; compared with the scrambled group, # P<0.05, ## P<0.01.

    Journal: Journal of Inflammation Research

    Article Title: TXNIP Participated in NLRP3-Mediated Inflammation in a Rat Model of Cervical Spondylotic Myelopathy

    doi: 10.2147/JIR.S373614

    Figure Lengend Snippet: Inhibition of TXNIP improved CSM-induced behavioral deficits. ( A ) The21-point Basso, Beattie and Bresnahan (BBB) locomotor rating scale; ( B ) cold allodynia; ( C ) inclined plane test. Compared with the sham group, **P<0.01; compared with the scrambled group, # P<0.05, ## P<0.01.

    Article Snippet: The primary antibodies included rabbit anti-rat TXNIP (1:800, MA5-32771, ThermoFisher), NLRP3 (1:800, MA5-32255, ThermoFisher), pro-caspase-1 (1:800, MA5-35833, ThermoFisher) and GAPDH (1:10000, PA1-16777, ThermoFisher).

    Techniques: Inhibition

    Inhibition of TXNIP reduced apoptosis in the anterior horn of the lesioned area following CSM. ( A ) Positive TUNEL staining control. ( B ) Apoptosis in the anterior horn of the lesioned area was analyzed by TUNEL staining. Black arrows show apoptotic cells. Apoptosis rate (%) = the numbers of apoptosis cells/total numbers of cells ×100%. The numbers of cells were counted using Image J software. Compared with the sham group, **P<0.01; compared with the scrambled group, ## P<0.01.

    Journal: Journal of Inflammation Research

    Article Title: TXNIP Participated in NLRP3-Mediated Inflammation in a Rat Model of Cervical Spondylotic Myelopathy

    doi: 10.2147/JIR.S373614

    Figure Lengend Snippet: Inhibition of TXNIP reduced apoptosis in the anterior horn of the lesioned area following CSM. ( A ) Positive TUNEL staining control. ( B ) Apoptosis in the anterior horn of the lesioned area was analyzed by TUNEL staining. Black arrows show apoptotic cells. Apoptosis rate (%) = the numbers of apoptosis cells/total numbers of cells ×100%. The numbers of cells were counted using Image J software. Compared with the sham group, **P<0.01; compared with the scrambled group, ## P<0.01.

    Article Snippet: The primary antibodies included rabbit anti-rat TXNIP (1:800, MA5-32771, ThermoFisher), NLRP3 (1:800, MA5-32255, ThermoFisher), pro-caspase-1 (1:800, MA5-35833, ThermoFisher) and GAPDH (1:10000, PA1-16777, ThermoFisher).

    Techniques: Inhibition, TUNEL Assay, Staining, Control, Software

    Inhibition of TXNIP declined the TXNIP expression in the anterior horn of the lesioned area following CSM. The mean fluorescence intensities of TXNIP/NeuN were analyzed by immunofluorescence. The mean gray values were analyzed using Image J software. Compared with the sham group, *P<0.05, **P<0.01; compared with the scrambled group, ## P<0.01.

    Journal: Journal of Inflammation Research

    Article Title: TXNIP Participated in NLRP3-Mediated Inflammation in a Rat Model of Cervical Spondylotic Myelopathy

    doi: 10.2147/JIR.S373614

    Figure Lengend Snippet: Inhibition of TXNIP declined the TXNIP expression in the anterior horn of the lesioned area following CSM. The mean fluorescence intensities of TXNIP/NeuN were analyzed by immunofluorescence. The mean gray values were analyzed using Image J software. Compared with the sham group, *P<0.05, **P<0.01; compared with the scrambled group, ## P<0.01.

    Article Snippet: The primary antibodies included rabbit anti-rat TXNIP (1:800, MA5-32771, ThermoFisher), NLRP3 (1:800, MA5-32255, ThermoFisher), pro-caspase-1 (1:800, MA5-35833, ThermoFisher) and GAPDH (1:10000, PA1-16777, ThermoFisher).

    Techniques: Inhibition, Expressing, Fluorescence, Immunofluorescence, Software

    Inhibition of TXNIP declined the NLRP3 expression in the anterior horn of the lesioned area following CSM. The mean fluorescence intensities of NLRP3/NeuN were analyzed by immunofluorescence. The mean gray values were analyzed by Image J software. Compared with the sham group, **P<0.01; compared with the scrambled group, ## P<0.01.

    Journal: Journal of Inflammation Research

    Article Title: TXNIP Participated in NLRP3-Mediated Inflammation in a Rat Model of Cervical Spondylotic Myelopathy

    doi: 10.2147/JIR.S373614

    Figure Lengend Snippet: Inhibition of TXNIP declined the NLRP3 expression in the anterior horn of the lesioned area following CSM. The mean fluorescence intensities of NLRP3/NeuN were analyzed by immunofluorescence. The mean gray values were analyzed by Image J software. Compared with the sham group, **P<0.01; compared with the scrambled group, ## P<0.01.

    Article Snippet: The primary antibodies included rabbit anti-rat TXNIP (1:800, MA5-32771, ThermoFisher), NLRP3 (1:800, MA5-32255, ThermoFisher), pro-caspase-1 (1:800, MA5-35833, ThermoFisher) and GAPDH (1:10000, PA1-16777, ThermoFisher).

    Techniques: Inhibition, Expressing, Fluorescence, Immunofluorescence, Software

    Inhibition TXNIP declined the NLRP3 mediated pro-caspase-1 expression in the anterior horn of the lesioned area following CSM. ( A ) The mean fluorescence intensities of pro-caspase-1/NeuN were analyzed by immunofluorescence. ( B ) The expression levels of TXNIP, NLRP3 and pro-caspase-1 in the lesioned spinal cords were measured by Western blot. The mean grays were analyzed by the Image J software. Compared with the sham group, **P<0.01; compared with the scrambled group, ## P<0.01.

    Journal: Journal of Inflammation Research

    Article Title: TXNIP Participated in NLRP3-Mediated Inflammation in a Rat Model of Cervical Spondylotic Myelopathy

    doi: 10.2147/JIR.S373614

    Figure Lengend Snippet: Inhibition TXNIP declined the NLRP3 mediated pro-caspase-1 expression in the anterior horn of the lesioned area following CSM. ( A ) The mean fluorescence intensities of pro-caspase-1/NeuN were analyzed by immunofluorescence. ( B ) The expression levels of TXNIP, NLRP3 and pro-caspase-1 in the lesioned spinal cords were measured by Western blot. The mean grays were analyzed by the Image J software. Compared with the sham group, **P<0.01; compared with the scrambled group, ## P<0.01.

    Article Snippet: The primary antibodies included rabbit anti-rat TXNIP (1:800, MA5-32771, ThermoFisher), NLRP3 (1:800, MA5-32255, ThermoFisher), pro-caspase-1 (1:800, MA5-35833, ThermoFisher) and GAPDH (1:10000, PA1-16777, ThermoFisher).

    Techniques: Inhibition, Expressing, Fluorescence, Immunofluorescence, Western Blot, Software

    The impact of ROS and NLRP3 inflammasome on the expression of pyroptosis associated gene and protein. (A)The gene and protein expression of TXNIP, NLRP3, and Caspae-1 in NRK-52E cells under 200 μg/ml uric acid stimulation or not for 24 hr detected by Real-time PCR and western blot techniques (n=3). The cells were categorized into four groups: the control group (0 μg/ml uric acid), the UA group (200 μg/ml uric), the UA+ROS (−) group (200 μg/ml uric + the ROS inhibitors NAC), as well as the UA+mtROS (−) group (200 μg/ml uric+ mtROS inhibitors mt-TEMPO). Data were expressed as mean ± SD. Compared with the UA group, ** P < 0.0001, * P < 0.05. Compared with the UA+ROS (−) group, ⋆⋆ P < 0.0001, ⋆ P < 0.05. Compared with UA+mtROS(−) group, # # P < 0.0001, # P < 0.05.(B) The pyroptosis percent in NRK-52E cells subjected to 200 μg/ml uric acid or not for 24 hr via flow cytometry method (n=3). The cells were divided into four groups: the control group (0 μg/ml uric acid), the UA group (200 μg/ml uric), the UA+NLRP3 (−) group (200 μg/ml uric + the NLRP3 inhibitors MCC950), the UA+Caspase-1(−) group (200 μg/ml uric+ Caspase-1 inhibitors Z-YVAD-FMK). Data were expressed as mean ± SD. Compared with the UA group, ** P < 0.0001, * P < 0.05. Compared with the UA+NLRP3 (-) group, ⋆⋆ P < 0.0001, ⋆ P < 0.05. Compared with UA+Caspase-1(−) group, ## P < 0.0001, # P < 0.05.(C) The gene and protein expression of NLRP3, ASC, Caspae-1, Activated-Caspase-1, GSDMD, IL-1β, and IL-18 in NRK-52E cells when exposed to 200 μg/ml uric acid or not for 24 hr using Real-time PCR and western blot method (n=3).

    Journal: bioRxiv

    Article Title: Hyperuricemia triggers Renal Tubular Epithelial Pyroptosis by using ROS to activate the NLRP3 inflammasome

    doi: 10.1101/2022.03.12.484115

    Figure Lengend Snippet: The impact of ROS and NLRP3 inflammasome on the expression of pyroptosis associated gene and protein. (A)The gene and protein expression of TXNIP, NLRP3, and Caspae-1 in NRK-52E cells under 200 μg/ml uric acid stimulation or not for 24 hr detected by Real-time PCR and western blot techniques (n=3). The cells were categorized into four groups: the control group (0 μg/ml uric acid), the UA group (200 μg/ml uric), the UA+ROS (−) group (200 μg/ml uric + the ROS inhibitors NAC), as well as the UA+mtROS (−) group (200 μg/ml uric+ mtROS inhibitors mt-TEMPO). Data were expressed as mean ± SD. Compared with the UA group, ** P < 0.0001, * P < 0.05. Compared with the UA+ROS (−) group, ⋆⋆ P < 0.0001, ⋆ P < 0.05. Compared with UA+mtROS(−) group, # # P < 0.0001, # P < 0.05.(B) The pyroptosis percent in NRK-52E cells subjected to 200 μg/ml uric acid or not for 24 hr via flow cytometry method (n=3). The cells were divided into four groups: the control group (0 μg/ml uric acid), the UA group (200 μg/ml uric), the UA+NLRP3 (−) group (200 μg/ml uric + the NLRP3 inhibitors MCC950), the UA+Caspase-1(−) group (200 μg/ml uric+ Caspase-1 inhibitors Z-YVAD-FMK). Data were expressed as mean ± SD. Compared with the UA group, ** P < 0.0001, * P < 0.05. Compared with the UA+NLRP3 (-) group, ⋆⋆ P < 0.0001, ⋆ P < 0.05. Compared with UA+Caspase-1(−) group, ## P < 0.0001, # P < 0.05.(C) The gene and protein expression of NLRP3, ASC, Caspae-1, Activated-Caspase-1, GSDMD, IL-1β, and IL-18 in NRK-52E cells when exposed to 200 μg/ml uric acid or not for 24 hr using Real-time PCR and western blot method (n=3).

    Article Snippet: Rabbit anti-rat TXNIP (ab188865), Caspase-1(ab108362), IL-18(ab191860), IL-1β (ab9722) , Caspase-1 p10 (ab179515) antibody were purchased from Abcam (Britain).

    Techniques: Expressing, Real-time Polymerase Chain Reaction, Western Blot, Flow Cytometry